dc.contributor.author |
Ramis, Ivy Bastos |
|
dc.contributor.author |
Fonseca, Tesiê Leopoldo |
|
dc.contributor.author |
Moraes, Ernani Pinho de |
|
dc.contributor.author |
Fernandes, Márcia Silveira |
|
dc.contributor.author |
Sassi, Raul Andrés Mendonza |
|
dc.contributor.author |
Rodrigues, Obirajara |
|
dc.contributor.author |
Juliano, Carlos Renan Varela |
|
dc.contributor.author |
Scaini, Carlos James |
|
dc.contributor.author |
Silva, Pedro Eduardo Almeida da |
|
dc.date.accessioned |
2012-09-17T22:20:27Z |
|
dc.date.available |
2012-09-17T22:20:27Z |
|
dc.date.issued |
2010 |
|
dc.identifier.citation |
RAMIS, Ivy Bastos et al. Molecular basis of the pathogenicity in helicobacter pylori clinical isolates. Journal of Clinical Microbiology, v. 48, n. 10, p. 3776–3778, 2010. Disponível em: <http://jcm.asm.org/content/48/10/3776.full.pdf+html>. Acesso em: 13 set. 2012. |
pt_BR |
dc.identifier.uri |
http://repositorio.furg.br/handle/1/2513 |
|
dc.description.abstract |
This study identified pathogenicity genes in 40 Helicobacter pylori clinical isolates. The cagA, vacA, and iceA genes were detected in 65%, 97.5%, and 97.5% of the isolates, respectively. The cagA, iceA1, and vacAs1a/m1 genes were related to erosive gastritis, whereas the vacAs2/m2 and iceA2 genes were associated with enanthematous gastritis. |
pt_BR |
dc.language.iso |
eng |
pt_BR |
dc.rights |
open access |
pt_BR |
dc.title |
Molecular basis of pathogenicity in helicobacter pylori clinical isolates |
pt_BR |
dc.type |
article |
pt_BR |